mTOR favors senescence over quiescence in p53-arrested cells.
Schug
Thaddeus T
TT
Laboratory of Signal Transduction, National Institute of Environmental Health Sciences, National Institutes of Health, Research Triangle Park, NC 27709, USA. Schugt@niehs.nih.gov
D000818:Animals; D016922:Cellular Senescence / Q000502:physiology*; D047908:Intracellular Signaling Peptides and Proteins / Q000378:metabolism*; D051379:Mice; D017346:Protein Serine-Threonine Kinases / Q000378:metabolism*; D015398:Signal Transduction / Q000502:physiology*; D058570:TOR Serine-Threonine Kinases; D016159:Tumor Suppressor Protein p53 / Q000378:metabolism*
D047908:Intracellular Signaling Peptides and Proteins; D016159:Tumor Suppressor Protein p53; C546843:mTOR protein, mouse; D017346:Protein Serine-Threonine Kinases; D058570:TOR Serine-Threonine Kinases