title: Wnt3a nonredundantly controls hematopoietic stem cell function and its deficiency results in complete absence of canonical Wnt signaling.
authors.forename: Tiago C
authors.initials: TC
authors.lastname: Luis
authors.forename: Brigitta A E
authors.initials: BA
authors.lastname: Naber
authors.forename: Willem E
authors.initials: WE
authors.lastname: Fibbe
authors.forename: Jacques J M
authors.initials: JJ
authors.lastname: van Dongen
authors.forename: Frank J T
authors.initials: FJ
authors.lastname: Staal
mesh_terms: D000818:Animals; D006410:Hematopoiesis; D006412:Hematopoietic Stem Cells / Q000502:physiology*; D008099:Liver / Q000166:cytology / Q000196:embryology; D051379:Mice; D008822:Mice, Transgenic; D015398:Signal Transduction; D051153:Wnt Proteins / Q000172:deficiency / Q000235:genetics / Q000502:physiology*; D060508:Wnt3 Protein; D060509:Wnt3A Protein
chemical_list: D051153:Wnt Proteins; D060508:Wnt3 Protein; D060509:Wnt3A Protein; C555511:Wnt3a protein, mouse