[A new view on pathochemical mechanisms of prolonged peritoneal dialysis].
New data on etiology, pathogenesis, clinics, quantity estimation, treatment and complications of peritoneal dialysis are observed. The role of aquaporine, nitric oxide, NO-synthase, inflammation and sepsis markers (procalcitonine, C-reactive protein) in pathochemical mechanism of peritoneal dialysis is discussed.
Petrovich
Iu A
IuA
Iarema
I V
IV
Terekhina
N A
NA
Kichenko
S M
SM
D000818:Animals; D020346:Aquaporins / Q000378:metabolism; D006801:Humans; D018836:Inflammation Mediators / Q000097:blood; D009569:Nitric Oxide / Q000097:blood; D019001:Nitric Oxide Synthase / Q000378:metabolism; D010530:Peritoneal Dialysis / Q000009:adverse effects*
D020346:Aquaporins; D018836:Inflammation Mediators; D009569:Nitric Oxide; D019001:Nitric Oxide Synthase