Blockade of receptor-activated G(i) signaling in osteoblasts in vivo leads to site-specific increases in cortical and cancellous bone formation.
Millard
Susan M
SM
Endocrine Research Unit, Veterans Administration Medical Center, University of California San Francisco, San Francisco, CA 94121, USA.
Louie
Alyssa M
AM
Wattanachanya
Lalita
L
Wronski
Thomas J
TJ
Conklin
Bruce R
BR
Nissenson
Robert A
RA