Excessive apoptosis as a downstream molecular event during the development of congenital heart diseases.
Kaichi
Shinji
S
Hasegawa
Koji
K
Morimoto
Tatsuya
T
D000199:Actins / Q000235:genetics / Q000378:metabolism; D000367:Age Factors; D000818:Animals; D017209:Apoptosis; D051017:Apoptosis Regulatory Proteins / Q000235:genetics / Q000378:metabolism; D015536:Down-Regulation; D018507:Gene Expression Regulation, Developmental; D005865:Gestational Age; D006330:Heart Defects, Congenital / Q000235:genetics / Q000378:metabolism / Q000473:pathology*; D006801:Humans; D032383:Myocytes, Cardiac / Q000378:metabolism / Q000473:pathology*; D012333:RNA, Messenger / Q000378:metabolism; D014157:Transcription Factors / Q000235:genetics / Q000378:metabolism
C526913:ACTC1 protein, human; D000199:Actins; D051017:Apoptosis Regulatory Proteins; D012333:RNA, Messenger; D014157:Transcription Factors