Secretion of MIP-1β and MIP-1α by CD8(+) T-lymphocytes correlates with HIV-1 inhibition independent of coreceptor usage.
Saunders
Kevin O
KO
Duke Human Vaccine Institute, Duke University Medical Center, Durham, NC 27710, USA. Kevin.saunders@duke.edu
Ward-Caviness
Cavin
C
Schutte
Robert J
RJ
Freel
Stephanie A
SA
Overman
R Glenn
RG
Thielman
Nathan M
NM
Cunningham
Coleen K
CK
Kepler
Thomas B
TB
Tomaras
Georgia D
GD
D000107:Acetylation; D018414:CD8-Positive T-Lymphocytes / Q000276:immunology*; D054405:Chemokine CCL3 / Q000378:metabolism*; D054407:Chemokine CCL4 / Q000378:metabolism*; D054357:Chemokine CXCL10 / Q000378:metabolism; D015536:Down-Regulation / Q000276:immunology; D015870:Gene Expression; D015658:HIV Infections / Q000276:immunology*; D015497:HIV-1 / Q000276:immunology*; D006657:Histones / Q000276:immunology; D006801:Humans; D007371:Interferon-gamma / Q000378:metabolism; D015854:Up-Regulation; D014635:Valproic Acid / Q000276:immunology; D014779:Virus Replication / Q000276:immunology
C515429:CCL3 protein, human; C066507:CCL4 protein, human; C114753:CXCL10 protein, human; D054405:Chemokine CCL3; D054407:Chemokine CCL4; D054357:Chemokine CXCL10; D006657:Histones; D014635:Valproic Acid; D007371:Interferon-gamma