Pilocytic astrocytomas have telomere-associated promyelocytic leukemia bodies without alternatively lengthened telomeres.
Slatter
Tania
T
Department of Pathology, Dunedin School of Medicine, PO Box 913, University of Otago, Dunedin, New Zealand.
Gifford-Garner
Jennifer
J
Wiles
Anna
A
Tan
Xin
X
Chen
Yu-Jen
YJ
MacFarlane
Martin
M
Sullivan
Michael
M
Royds
Janice
J
Hung
Noelyn
N
D000293:Adolescent; D000328:Adult; D001254:Astrocytoma / Q000235:genetics* / Q000378:metabolism* / Q000473:pathology; D001932:Brain Neoplasms / Q000235:genetics* / Q000378:metabolism* / Q000473:pathology; D002648:Child; D018450:Disease Progression; D006801:Humans; D002479:Inclusion Bodies / Q000378:metabolism; D008875:Middle Aged; D009367:Neoplasm Staging; D009687:Nuclear Proteins / Q000378:metabolism*; D000071156:Promyelocytic Leukemia Protein; D016615:Telomere / Q000737:chemistry / Q000235:genetics / Q000378:metabolism*; D014157:Transcription Factors / Q000378:metabolism*; D025521:Tumor Suppressor Proteins / Q000378:metabolism*; D055815:Young Adult
D009687:Nuclear Proteins; D000071156:Promyelocytic Leukemia Protein; D014157:Transcription Factors; D025521:Tumor Suppressor Proteins; C070576:PML protein, human