The effects of dexamethasone, ascorbic acid, and β-glycerophosphate on osteoblastic differentiation by regulating estrogen receptor and osteopontin expression.
Park
Jun-Beom
JB
Department of Molecular Medicine and Biopharmaceutical Sciences, Graduate School of Convergence Science and Technology, Seoul National University, Seoul, Korea. jbassoonis@yahoo.co.kr
D000469:Alkaline Phosphatase / Q000378:metabolism; D000818:Animals; D001205:Ascorbic Acid / Q000494:pharmacology*; D002113:Calcification, Physiologic / Q000187:drug effects; D002454:Cell Differentiation / Q000187:drug effects*; D002460:Cell Line; D049109:Cell Proliferation / Q000187:drug effects; D002470:Cell Survival / Q000187:drug effects; D002478:Cells, Cultured; D003907:Dexamethasone / Q000494:pharmacology*; D004305:Dose-Response Relationship, Drug; D005994:Glycerophosphates / Q000494:pharmacology*; D051379:Mice; D023421:Models, Animal; D010006:Osteoblasts / Q000166:cytology* / Q000187:drug effects / Q000378:metabolism*; D053495:Osteopontin / Q000378:metabolism*; D010641:Phenotype; D011960:Receptors, Estrogen / Q000378:metabolism*; D016212:Transforming Growth Factor beta / Q000378:metabolism
D005994:Glycerophosphates; D011960:Receptors, Estrogen; D016212:Transforming Growth Factor beta; D053495:Osteopontin; D003907:Dexamethasone; D000469:Alkaline Phosphatase; D001205:Ascorbic Acid; C031463:beta-glycerophosphoric acid