Hyaluronan fragments contribute to the ozone-primed immune response to lipopolysaccharide.
Li
Zhuowei
Z
Division of Pulmonary, Allergy, and Critical Care Medicine, Department of Medicine, Duke University, Durham, NC 27710, USA.
Potts
Erin N
EN
Piantadosi
Claude A
CA
Foster
W Michael
WM
Hollingsworth
John W
JW
D055371:Acute Lung Injury / Q000139:chemically induced / Q000276:immunology / Q000473:pathology; D000280:Administration, Inhalation; D000818:Animals; D001854:Bone Marrow Cells / Q000187:drug effects / Q000276:immunology / Q000378:metabolism; D001992:Bronchoalveolar Lavage Fluid / Q000737:chemistry / Q000166:cytology / Q000276:immunology; D002478:Cells, Cultured; D004306:Dose-Response Relationship, Immunologic; D018960:Hyaluronan Receptors / Q000378:metabolism; D006820:Hyaluronic Acid / Q000096:biosynthesis / Q000737:chemistry / Q000633:toxicity*; D008070:Lipopolysaccharides / Q000008:administration & dosage* / Q000737:chemistry / Q000494:pharmacology; D016676:Macrophages, Alveolar / Q000187:drug effects / Q000276:immunology / Q000378:metabolism; D008297:Male; D051379:Mice; D008810:Mice, Inbred C57BL; D008970:Molecular Weight; D010126:Ozone / Q000008:administration & dosage / Q000737:chemistry / Q000633:toxicity*
D018960:Hyaluronan Receptors; D008070:Lipopolysaccharides; D010126:Ozone; D006820:Hyaluronic Acid