Dermal fibroblasts from acute inflamed atopic dermatitis lesions display increased eotaxin/CCL11 responsiveness to interleukin-4 stimulation.
Gahr
N
N
Clinical Research Unit, Department of Dermatology, University Medical Center Schleswig-Holstein, Campus Kiel, Germany.
Fölster-Holst
R
R
Weichenthal
M
M
Christophers
E
E
Schröder
J-M
JM
Bartels
J
J
D000208:Acute Disease; D000328:Adult; D000368:Aged; D001706:Biopsy; D002478:Cells, Cultured; D054413:Chemokine CCL11 / Q000378:metabolism*; D002648:Child; D002908:Chronic Disease; D003876:Dermatitis, Atopic / Q000378:metabolism*; D004305:Dose-Response Relationship, Drug; D005260:Female; D005347:Fibroblasts / Q000187:drug effects* / Q000378:metabolism; D006801:Humans; D015847:Interleukin-4 / Q000494:pharmacology*; D008297:Male; D008875:Middle Aged; D012333:RNA, Messenger / Q000378:metabolism; D020133:Reverse Transcriptase Polymerase Chain Reaction / Q000379:methods
D054413:Chemokine CCL11; D012333:RNA, Messenger; D015847:Interleukin-4