Sox2 expression is maintained while gastric phenotype is completely lost in Cdx2-induced intestinal metaplastic mucosa.
Mutoh
Hiroyuki
H
Department of Medicine, Division of Gastroenterology, Jichi Medical University, 3311-1 Yakushiji, Shimotsuke, Tochigi 329-0498, Japan. muto@jichi.ac.jp
Sashikawa
Miho
M
Sugano
Kentaro
K
D000469:Alkaline Phosphatase / Q000235:genetics / Q000378:metabolism; D000818:Animals; D000071616:CDX2 Transcription Factor; D002460:Cell Line; D005753:Gastric Mucosa / Q000382:microbiology / Q000473:pathology*; D020869:Gene Expression Profiling; D016480:Helicobacter pylori; D018398:Homeodomain Proteins / Q000235:genetics / Q000378:metabolism*; D006801:Humans; D007413:Intestinal Mucosa / Q000382:microbiology / Q000473:pathology*; D008679:Metaplasia / Q000235:genetics / Q000382:microbiology / Q000473:pathology; D051379:Mice; D008822:Mice, Transgenic; D055271:Mucin 5AC / Q000235:genetics / Q000378:metabolism*; D010641:Phenotype; D055748:SOXB1 Transcription Factors / Q000235:genetics / Q000378:metabolism*; D014157:Transcription Factors / Q000235:genetics / Q000378:metabolism*
D000071616:CDX2 Transcription Factor; C487514:Cdx2 protein, mouse; D018398:Homeodomain Proteins; C524679:Muc5ac protein, mouse; D055271:Mucin 5AC; D055748:SOXB1 Transcription Factors; C527783:Sox2 protein, mouse; D014157:Transcription Factors; D000469:Alkaline Phosphatase