Transcription factor Ap-1 mediates proangiogenic MIF expression in human endothelial cells exposed to Angiotensin II.
Shan
Zhi-Xin
ZX
Research Center of Guangdong General Hospital, Guangdong Provincial Cardiovascular Institute, Guangdong Academy of Medical Sciences, Guangzhou 510080, China.
Lin
Qiu-Xiong
QX
Yang
Min
M
Zhang
Bin
B
Zhu
Jie-Ning
JN
Mai
Li-Ping
LP
Deng
Chun-Yu
CY
Liu
Ju-Li
JL
Zhang
You-Yi
YY
Lin
Shu-Guang
SG
Yu
Xi-Yong
XY
D043925:Angiogenesis Inducing Agents / Q000378:metabolism*; D000804:Angiotensin II / Q000494:pharmacology*; D000818:Animals; D001483:Base Sequence; D002457:Cell Extracts; D002467:Cell Nucleus / Q000187:drug effects / Q000378:metabolism; D024202:Electrophoretic Mobility Shift Assay; D042783:Endothelial Cells / Q000187:drug effects* / Q000378:metabolism*; D006801:Humans; D008263:Macrophage Migration-Inhibitory Factors / Q000235:genetics / Q000378:metabolism* / Q000494:pharmacology; D008969:Molecular Sequence Data; D018919:Neovascularization, Physiologic / Q000187:drug effects*; D051381:Rats; D018808:Transcription Factor AP-1 / Q000378:metabolism*; D014471:Umbilical Veins / Q000166:cytology
D043925:Angiogenesis Inducing Agents; D002457:Cell Extracts; D008263:Macrophage Migration-Inhibitory Factors; D018808:Transcription Factor AP-1; D000804:Angiotensin II