title: Chronic formoterol administration reduces cardiac mitochondrial protein synthesis and oxidative capacity in mice.
authors.forename: Bertrand
authors.initials: B
authors.lastname: Léger
authors.forename: René
authors.initials: R
authors.lastname: Koopman
authors.forename: Stéphane
authors.initials: S
authors.lastname: Walrand
authors.forename: Stefan M
authors.initials: SM
authors.lastname: Gehrig
authors.forename: Kate T
authors.initials: KT
authors.lastname: Murphy
authors.forename: Gordon S
authors.initials: GS
authors.lastname: Lynch
mesh_terms: D058666:Adrenergic beta-2 Receptor Agonists / Q000494:pharmacology*; D000818:Animals; D002950:Citrate (si)-Synthase / Q000378:metabolism; D004983:Ethanolamines / Q000494:pharmacology*; D000068759:Formoterol Fumarate; D008297:Male; D051379:Mice; D008810:Mice, Inbred C57BL; D008928:Mitochondria / Q000187:drug effects* / Q000378:metabolism*; D009206:Myocardium / Q000378:metabolism*; D010084:Oxidation-Reduction / Q000187:drug effects
chemical_list: D058666:Adrenergic beta-2 Receptor Agonists; D004983:Ethanolamines; D002950:Citrate (si)-Synthase; D000068759:Formoterol Fumarate