title: Reversible cardiomyopathies--a review.
abstract: End-stage renal disease, cirrhosis, obesity, tachycardia, and extreme stress have all been shown to result in impaired left ventricular function. It is becoming clear, however, that the cardiomyopathies associated with these states are reversible after resolution of the underlying process. In this article, we present the current data demonstrating that renal transplantation, liver transplantation, and bariatric surgery can lead to reversal of uremic, cirrhotic, and obesity cardiomyopathies, respectively. We also discuss the reversibility of tachycardia-induced cardiomyopathy after radiofrequency ablation or pharmacologic therapy for rate or rhythm control and the reversibility of stress-induced cardiomyopathy with supportive care.
authors.affiliation: Department of Internal Medicine, Mount Sinai Hospital, New York, NY 10029, USA. christopher.j.huffman@gmail.com
authors.forename: C
authors.initials: C
authors.lastname: Huffman
authors.forename: G
authors.initials: G
authors.lastname: Wagman
authors.forename: M
authors.initials: M
authors.lastname: Fudim
authors.forename: R
authors.initials: R
authors.lastname: Zolty
authors.forename: T
authors.initials: T
authors.lastname: Vittorio
mesh_terms: D000818:Animals; D050110:Bariatric Surgery; D009202:Cardiomyopathies / Q000209:etiology / Q000503:physiopathology / Q000628:therapy*; D017115:Catheter Ablation; D006801:Humans; D007676:Kidney Failure, Chronic / Q000150:complications / Q000503:physiopathology / Q000601:surgery; D016030:Kidney Transplantation; D008103:Liver Cirrhosis / Q000150:complications / Q000503:physiopathology / Q000601:surgery; D016031:Liver Transplantation; D009765:Obesity / Q000150:complications / Q000503:physiopathology / Q000601:surgery; D020127:Recovery of Function; D013610:Tachycardia / Q000150:complications / Q000503:physiopathology / Q000601:surgery; D054549:Takotsubo Cardiomyopathy / Q000209:etiology / Q000503:physiopathology / Q000628:therapy*; D016896:Treatment Outcome; D018487:Ventricular Dysfunction, Left / Q000209:etiology / Q000503:physiopathology / Q000628:therapy*; D016277:Ventricular Function, Left