Histamine modulates the responsiveness of keratinocytes to IL-17 and TNF-α through the H1-receptor.
Catharina Sagita
CS
Moniaga
Gyohei
G
Egawa
Hiromi
H
Doi
Yoshiki
Y
Miyachi
Kenji
K
Kabashima
D002090:Butyrophenones / Q000494:pharmacology; D002478:Cells, Cultured; D016178:Granulocyte-Macrophage Colony-Stimulating Factor / Q000378:metabolism; D006632:Histamine / Q000502:physiology*; D006634:Histamine H1 Antagonists / Q000494:pharmacology; D006801:Humans; D007249:Inflammation / Q000503:physiopathology; D018836:Inflammation Mediators / Q000378:metabolism* / Q000502:physiology; D020381:Interleukin-17 / Q000502:physiology*; D016209:Interleukin-8 / Q000378:metabolism; D015603:Keratinocytes / Q000276:immunology / Q000378:metabolism* / Q000502:physiology; D010880:Piperidines / Q000494:pharmacology; D011969:Receptors, Histamine H1 / Q000187:drug effects / Q000502:physiology*; D014409:Tumor Necrosis Factor-alpha / Q000502:physiology*
D002090:Butyrophenones; D006634:Histamine H1 Antagonists; D018836:Inflammation Mediators; D020381:Interleukin-17; D016209:Interleukin-8; D010880:Piperidines; D011969:Receptors, Histamine H1; D014409:Tumor Necrosis Factor-alpha; C058251:carebastine; D006632:Histamine; D016178:Granulocyte-Macrophage Colony-Stimulating Factor