title: Cerebral hyperperfusion syndrome associated with non-convulsive status epilepticus following superficial temporal artery-middle cerebral artery anastomosis. Case report.
abstract: A 77-year-old man developed cerebral hyperperfusion syndrome with temporal deterioration of consciousness and worsening of left hemiparesis on the 6(th) postoperative day following superficial temporal artery-middle cerebral artery (STA-MCA) anastomosis for right M(1) occlusion. Electroencephalography (EEG) revealed frequent ictal discharges in the right hemisphere, although convulsive seizures were not apparent. Administration of anticonvulsants was performed based on the diagnosis of non-convulsive status epilepticus (NCSE). Complete recovery from hyperperfusion syndrome was achieved with rapid improvement of EEG findings. The present case demonstrates the pathophysiological mechanism of hyperperfusion syndrome associated with NCSE after STA-MCA anastomosis.
authors.affiliation: Department of Neurosurgery, Kyushu Rosai Hospital, Kitakyushu, Fukuoka.
authors.forename: Takeshi
authors.initials: T
authors.lastname: Hamamura
authors.forename: Takato
authors.initials: T
authors.lastname: Morioka
authors.forename: Tetsuro
authors.initials: T
authors.lastname: Sayama
authors.forename: Nobutaka
authors.initials: N
authors.lastname: Mukae
authors.forename: Shuji
authors.initials: S
authors.lastname: Arakawa
authors.forename: Hironobu
authors.initials: H
authors.lastname: Maeda
authors.forename: Tomio
authors.initials: T
authors.lastname: Sasaki
mesh_terms: D000368:Aged; D000714:Anastomosis, Surgical; D001783:Blood Flow Velocity; D001921:Brain / Q000098:blood supply* / Q000503:physiopathology; D002533:Cerebral Angiography; D002548:Cerebral Revascularization / Q000009:adverse effects*; D002560:Cerebrovascular Circulation; D004569:Electroencephalography; D006801:Humans; D002537:Intracranial Arteriosclerosis / Q000150:complications / Q000601:surgery*; D008297:Male; D020768:Middle Cerebral Artery / Q000601:surgery*; D013226:Status Epilepticus / Q000209:etiology* / Q000517:prevention & control; D013699:Temporal Arteries / Q000601:surgery*