title: [Meaning of the calcium regulating system in the pathogenesis of experimental gastric ulcers in rabbits].
abstract: The arm of the study was to examine the influence parathyrin and calcitonin on the development experimental gastric ulcers of rabbits.
  
  
  METHODS
  The study involved 87 rabbits. All animals were divided on six groups in dependence on experimental influence (control, trauma celiac plexus, application parathyrin, trauma celiac plexus and introduction parathyrin, application calcitonin, trauma celiac plexus and introduction calcitonin).
  
  
  CONCLUSIONS
  Experimental gastric ulcers reproducing of trauma celiac plexus are formed on the background of dysfunction parathyroid glands and C-cell thyroid gland and hypercalcemia. Parathyrin intensifies mechanisms of ulcer's formation, calcitonin prevents from it.
authors.forename: V V
authors.initials: VV
authors.lastname: Chernin
authors.forename: L A
authors.initials: LA
authors.lastname: Fomina
authors.forename: G V
authors.initials: GV
authors.lastname: Khomullo
mesh_terms: D000818:Animals; D050071:Bone Density Conservation Agents / Q000378:metabolism / Q000494:pharmacology; D002116:Calcitonin / Q000378:metabolism* / Q000494:pharmacology; D002118:Calcium / Q000378:metabolism*; D004195:Disease Models, Animal; D006934:Hypercalcemia; D010280:Parathyroid Glands / Q000378:metabolism* / Q000473:pathology; D010281:Parathyroid Hormone / Q000378:metabolism* / Q000494:pharmacology; D011817:Rabbits; D013276:Stomach Ulcer / Q000378:metabolism* / Q000473:pathology
chemical_list: D050071:Bone Density Conservation Agents; D010281:Parathyroid Hormone; D002116:Calcitonin; D002118:Calcium