title: [The resistive and capacity functions of the subcutaneous vein under blockade of calcium channels and endothelial factors].
abstract: In cats, the calcium antagonist Verapamil depressed vein constrictor responses to noradrenaline and angiotensine II both under conditions of wholesome endothelium and physical-chemical and chemical de-endothelisation. Following the de-endothelisation the vein responses recovered. The data obtained suggest that this recovery seems to be due to opening of new calcium channels that are independent of the Verapamil effect.
authors.forename: Iu A
authors.initials: IuA
authors.lastname: Kudriashov
authors.forename: T Iu
authors.initials: TIu
authors.lastname: Tsviashchenko
mesh_terms: D000804:Angiotensin II / Q000494:pharmacology; D000818:Animals; D002118:Calcium / Q000037:antagonists & inhibitors; D002121:Calcium Channel Blockers / Q000494:pharmacology*; D002415:Cats; D004730:Endothelium, Vascular / Q000502:physiology*; D009638:Norepinephrine / Q000494:pharmacology; D014655:Vascular Resistance / Q000187:drug effects* / Q000502:physiology; D014661:Vasoconstriction / Q000187:drug effects*; D014662:Vasoconstrictor Agents / Q000494:pharmacology; D014680:Veins / Q000502:physiology*; D014700:Verapamil / Q000494:pharmacology*
chemical_list: D002121:Calcium Channel Blockers; D014662:Vasoconstrictor Agents; D000804:Angiotensin II; D014700:Verapamil; D002118:Calcium; D009638:Norepinephrine