title: Familial or genetic primary aldosteronism and Gordon syndrome.
abstract: Salt-sensitive forms of hypertension have received considerable renewed attention in recent years. This article focuses on 2 main forms of salt-sensitive hypertension (familial or genetic primary aldosteronism [PA] and Gordon syndrome) and the current state of knowledge regarding their genetic bases. The glucocorticoid-remediable form of familial PA (familial hyperaldosteronism type I) is dealt with only briefly because it is covered in depth elsewhere.
authors.affiliation: Endocrine Hypertension Research Center, University of Queensland School of Medicine, Princess Alexandra Hospital, Ipswich Road, Woolloongabba, Brisbane 4102, Australia. m.stowasser@uq.edu.au
authors.forename: Michael
authors.initials: M
authors.lastname: Stowasser
authors.forename: Eduardo
authors.initials: E
authors.lastname: Pimenta
authors.forename: Richard D
authors.initials: RD
authors.lastname: Gordon
mesh_terms: D000818:Animals; D001176:Arthrogryposis / Q000175:diagnosis / Q000235:genetics / Q000503:physiopathology / Q000628:therapy; D002972:Cleft Palate / Q000175:diagnosis / Q000235:genetics / Q000503:physiopathology / Q000628:therapy; D003025:Clubfoot / Q000175:diagnosis / Q000235:genetics / Q000503:physiopathology / Q000628:therapy; D003937:Diagnosis, Differential; D004195:Disease Models, Animal; D014644:Genetic Variation; D006228:Hand Deformities, Congenital / Q000175:diagnosis / Q000235:genetics / Q000503:physiopathology / Q000628:therapy; D006801:Humans; D006929:Hyperaldosteronism / Q000175:diagnosis / Q000235:genetics* / Q000503:physiopathology* / Q000628:therapy; D008094:Lithium / Q000493:pharmacokinetics; D008657:Metabolic Clearance Rate; D017673:Sodium Chloride, Dietary / Q000009:adverse effects
chemical_list: D017673:Sodium Chloride, Dietary; D008094:Lithium