title: Maximisation of maternal cardiac output during labour might help to prevent not only foetal hypoxaemia but also myometrial ischaemia, dysfunctional labour, uterine atony and postpartum endometritis.
authors.forename: T L
authors.initials: TL
authors.lastname: Archer
authors.forename: A E
authors.initials: AE
authors.lastname: Shapiro
authors.forename: P J
authors.initials: PJ
authors.lastname: Suresh
mesh_terms: D000328:Adult; D002302:Cardiac Output / Q000502:physiology*; D004716:Endometritis / Q000517:prevention & control*; D005260:Female; D005311:Fetal Hypoxia / Q000517:prevention & control*; D006801:Humans; D007231:Infant, Newborn; D007511:Ischemia / Q000517:prevention & control*; D007743:Labor, Obstetric / Q000502:physiology*; D009215:Myometrium / Q000098:blood supply*; D009765:Obesity / Q000150:complications; D007744:Obstetric Labor Complications / Q000517:prevention & control*; D006831:Polyhydramnios / Q000503:physiopathology; D011247:Pregnancy; D016896:Treatment Outcome; D014599:Uterus / Q000502:physiology*