title: [Trigger mechanism of neuroprotective effect of ischemic preconditioning].
abstract: Analysis of literature data indicates that adenosine, glutamate, NO, reactive oxygen species, erythropoietin, interleukin-1 are triggers of delayed preconditioning of brain. The issue of involvement of bradykinin, opioids, TNF-alpha in preconditioning remains unclear, although possibility of these compounds to induce a state of increased tolerance to ischemia does not induce a doubt. It was established that adenosine receptor agonists, bradykinin, opioids, NO* donors can mimic a phenomenon of early preconditioning.
authors.forename: L N
authors.initials: LN
authors.lastname: Maslov
mesh_terms: D000241:Adenosine / Q000378:metabolism; D000818:Animals; D001920:Bradykinin / Q000378:metabolism; D001921:Brain / Q000098:blood supply* / Q000378:metabolism*; D016207:Cytokines / Q000378:metabolism; D005973:Glutamine / Q000378:metabolism; D006801:Humans; D019194:Ischemic Preconditioning; D009569:Nitric Oxide / Q000378:metabolism
chemical_list: D016207:Cytokines; D005973:Glutamine; D009569:Nitric Oxide; D000241:Adenosine; D001920:Bradykinin