title: [Comparative characteristic of energy supply disturbances in arterial and venous walls of rabbits with alloxan diabetes and monoiodacetate intoxication].
abstract: We showed here that energy metabolism in thoracic rabbit aorta and posterior vena cava is disturbed two weeks following the induction of alloxan-induced diabetes and monoiodacetate intoxication. In the vessels studied, the alterations are manifested in the decrease in the intensity of glucose uptake, oxygen consumption, lactic acid production, and ATP resynthesis. Simultaneously, the ATP content is significantly reduced. The possible significance of these disorders in the development of Monckeberg's arteriosclerosis is discussed.
authors.forename: O V
authors.initials: OV
authors.lastname: Ataman
authors.forename: Iu O
authors.initials: IuO
authors.lastname: Ataman
mesh_terms: D000255:Adenosine Triphosphate / Q000378:metabolism; D000496:Alloxan; D000818:Animals; D001013:Aorta, Thoracic / Q000187:drug effects / Q000378:metabolism*; D003921:Diabetes Mellitus, Experimental / Q000378:metabolism* / Q000503:physiopathology; D004734:Energy Metabolism / Q000187:drug effects*; D005947:Glucose / Q000378:metabolism; D019807:Iodoacetic Acid / Q000633:toxicity*; D019344:Lactic Acid / Q000378:metabolism; D008297:Male; D050380:Monckeberg Medial Calcific Sclerosis / Q000378:metabolism; D010101:Oxygen Consumption / Q000187:drug effects; D011817:Rabbits; D014684:Venae Cavae / Q000187:drug effects / Q000378:metabolism*
chemical_list: D019344:Lactic Acid; D000496:Alloxan; D000255:Adenosine Triphosphate; D005947:Glucose; D019807:Iodoacetic Acid