title: [Effect of nitric oxide/endothelin interaction on hyperoxic vasoconstruction].
abstract: The data obtained demonstrated that NO restrains ET-1 production and blunts ET-1-mediated basal cerebrovascular tone. Local hyperoxygenation of the brain tissue decreases NO availability, supeoxide production, suppresses NO-mediated vascular tone and facilitates ET-1-mediated vasoconstriction.
authors.forename: I T
authors.initials: IT
authors.lastname: Demchenko
authors.forename: A N
authors.initials: AN
authors.lastname: Moskvin
authors.forename: S Iu
authors.initials: SIu
authors.lastname: Zhiliaev
authors.forename: O S
authors.initials: OS
authors.lastname: Alekseeva
authors.forename: T Iu
authors.initials: TIu
authors.lastname: Postnikova
authors.forename: A I
authors.initials: AI
authors.lastname: Krivchenko
mesh_terms: D000818:Animals; D001921:Brain / Q000098:blood supply*; D016232:Endothelins / Q000502:physiology*; D018496:Hyperoxia / Q000503:physiopathology*; D008297:Male; D009569:Nitric Oxide / Q000502:physiology*; D051381:Rats; D017208:Rats, Wistar; D014661:Vasoconstriction / Q000502:physiology*
chemical_list: D016232:Endothelins; D009569:Nitric Oxide