title: HIV infection and clinical spectrum of associated vasculitides.
abstract: Various infections have been causative in the pathogenesis of systemic vasculitides, and HIV infection is not spared. In an immunocompromised host, cytomegalovirus, Epstein-Barr virus, varicella zoster virus, herpes simplex virus, hepatitis B and hepatitis C virus, and mycobacteria, along with HIV infection can cause vasculitis. Herein we emphasize the spectrum of vasculitides, their pathogenesis, presentation, course, and therapy in the HIV-infected population. Every spectrum and size of the blood vessel involvement have been seen in HIV-associated vasculitides. We review each spectrum in detail and describe our experience with polyarteritis nodosa, the most common presentation occurring in HIV-infected patients. We also discuss the differences in HIV, hepatitis B, and hepatitis C- related polyarteritis nodosa in detail.
authors: Section of Rheumatology, Louisiana State University Health Sciences Center, New Orleans, LA 70112, USA. npate5@lsuhsc.edu
authors: Nirupa
authors: N
authors: Patel
authors: Neema
authors: N
authors: Patel
authors: Tahir
authors: T
authors: Khan
authors: Neej
authors: N
authors: Patel
authors: Luis R
authors: LR
authors: Espinoza
mesh_terms: D017088:AIDS-Related Opportunistic Infections / Q000276:immunology / Q000473:pathology; D044966:Anti-Retroviral Agents / Q000627:therapeutic use; D015984:Causality; D005938:Glucocorticoids / Q000627:therapeutic use; D015658:HIV Infections / Q000188:drug therapy / Q000276:immunology / Q000473:pathology*; D006509:Hepatitis B / Q000276:immunology / Q000473:pathology; D006526:Hepatitis C / Q000276:immunology / Q000473:pathology; D006801:Humans; D016867:Immunocompromised Host; D010488:Polyarteritis Nodosa / Q000188:drug therapy / Q000473:pathology / Q000821:virology; D056647:Systemic Vasculitis / Q000188:drug therapy / Q000473:pathology* / Q000821:virology
chemical_list: D044966:Anti-Retroviral Agents; D005938:Glucocorticoids