title: Do reactive oxygen species regulate skeletal muscle glucose uptake during contraction?
abstract: The mechanisms regulating skeletal muscle glucose uptake during contraction remain poorly defined. Recent studies suggest reactive oxygen species may be an important signaling intermediate for the regulation of skeletal muscle glucose uptake during ex vivo contraction but less important during more physiological conditions, such as in situ muscle contraction in rodents and during exercise in humans.
authors.affiliation: Department of Biochemistry and Molecular Biology, Monash University, Clayton, Victoria 3800, Australia. troy.merry@monash.edu
authors.forename: Troy L
authors.initials: TL
authors.lastname: Merry
authors.forename: Glenn K
authors.initials: GK
authors.lastname: McConell
mesh_terms: D000818:Animals; D001692:Biological Transport; D015444:Exercise / Q000502:physiology; D005947:Glucose / Q000378:metabolism*; D006801:Humans; D009119:Muscle Contraction / Q000502:physiology*; D018482:Muscle, Skeletal / Q000378:metabolism* / Q000502:physiology; D009569:Nitric Oxide / Q000378:metabolism; D011494:Protein Kinases / Q000378:metabolism; D017382:Reactive Oxygen Species / Q000378:metabolism*; D015398:Signal Transduction / Q000502:physiology
chemical_list: D017382:Reactive Oxygen Species; D009569:Nitric Oxide; D011494:Protein Kinases; D005947:Glucose