[Signal transduction pathways involved in progression to androgen-independent prostate cancer].
Department of Urology, Kyoto University Graduate School of Medicine.
Takahiro
T
Inoue
D000728:Androgens / Q000502:physiology*; D000818:Animals; D006801:Humans; D008297:Male; D015514:Oncogene Proteins, Fusion / Q000235:genetics; D051059:PTEN Phosphohydrolase / Q000235:genetics; D011471:Prostatic Neoplasms / Q000503:physiopathology*; D011493:Protein Kinase C / Q000502:physiology; D011944:Receptors, Androgen / Q000502:physiology; D058309:Receptors, Prostaglandin E, EP4 Subtype / Q000502:physiology; D019893:Ribosomal Protein S6 Kinases / Q000502:physiology; D015398:Signal Transduction / Q000502:physiology*; D058570:TOR Serine-Threonine Kinases / Q000502:physiology; D020830:rac1 GTP-Binding Protein / Q000502:physiology
D000728:Androgens; D015514:Oncogene Proteins, Fusion; C107953:RAC1 protein, human; D011944:Receptors, Androgen; D058309:Receptors, Prostaglandin E, EP4 Subtype; C515553:TMPRSS2-ERG fusion protein, human; D019893:Ribosomal Protein S6 Kinases; D058570:TOR Serine-Threonine Kinases; C115015:PKC-3 protein; D011493:Protein Kinase C; D051059:PTEN Phosphohydrolase; D020830:rac1 GTP-Binding Protein