title: [Non-tumor bile duct strictures].
abstract: The authors rectrospectively evaluate the possibilities of the solutions and the complications regarding benign stenoses of the biliary tract in 10 patients, who were treated within the years 2008 - 4/2011 at the Surgical Clinic of Medical Faculty of Charles University and the Faculty Hospital in Plzen. The injury of the biliary tree at cholecystectomy - 6 cases - was the most often cause of the benign stenosis of the biliary tree development in our collection. Once it was the case of a rare incidence of M. Crohn's disease of the biliary tract, once it was the case of stenosis in Mirrizi syndrome and two times it was the case of stenosis after the repeated instrumental procedures at ERCP after choledocholithiasis. Three times stenosis was solved by help of the repeated dilations of stenosis in the way of ERCP procedure. In other 7 cases stenosis was solved surgically by help of hepaticojejunalanastomosis to the excluded Roux-en-Y gastric bypass. The dilation of stenosis of the biliary tree requested two sessions on average, the surgical solution had a minimal peri-operative morbidity - 0% and within a 30-day peri-operative period there was noted 1 death in connection with the solution of stenosis of the biliary tract with the massive bleeding at decubitus of the stent of the left biliary duct into hepatic artery. Long-term results with respect to a short-time period were not evaluated.
authors.affiliation: Chirurgická klinika LF UK a FN v Plzni. skalicky@fnplzen.cz
authors.forename: T
authors.initials: T
authors.lastname: Skalický
authors.forename: V
authors.initials: V
authors.lastname: Treska
authors.forename: A
authors.initials: A
authors.lastname: Sutnar
authors.forename: V
authors.initials: V
authors.lastname: Liska
authors.forename: P
authors.initials: P
authors.lastname: Duras
authors.forename: F
authors.initials: F
authors.lastname: Slauf
authors.forename: J
authors.initials: J
authors.lastname: Kozeluhová
mesh_terms: D002779:Cholestasis / Q000175:diagnosis / Q000209:etiology* / Q000628:therapy; D006801:Humans