title: The influence of triamcinolone on endostatin-like proteins in oxygen-induced retinopathy of prematurity.
abstract: In the murine model of oxygen-induced retinopathy of prematurity, early treatment (right after oxygen exposure) with triamcinolone reduced neovascularization and subsequently endostatin presence. Late treatment (3 days after oxygen exposure) limited pathological vascular sprouting, not interfering with normal vascularization of the retina. The effects remained unilateral in the treated eyes.
authors.forename: Christian Albrecht
authors.initials: CA
authors.lastname: May
mesh_terms: D020533:Angiogenesis Inhibitors / Q000378:metabolism*; D000818:Animals; D004195:Disease Models, Animal; D043169:Endostatins / Q000378:metabolism*; D005938:Glucocorticoids / Q000008:administration & dosage*; D006801:Humans; D007231:Infant, Newborn; D058449:Intravitreal Injections; D051379:Mice; D010100:Oxygen / Q000633:toxicity; D015861:Retinal Neovascularization / Q000378:metabolism / Q000473:pathology / Q000517:prevention & control*; D012171:Retinal Vessels / Q000473:pathology; D012178:Retinopathy of Prematurity / Q000188:drug therapy* / Q000378:metabolism / Q000473:pathology; D014222:Triamcinolone Acetonide / Q000008:administration & dosage*
chemical_list: D020533:Angiogenesis Inhibitors; D043169:Endostatins; D005938:Glucocorticoids; D014222:Triamcinolone Acetonide; D010100:Oxygen