Hypoxia favors maintenance of the vascular smooth muscle cell phenotype in culture.
Department of Orthopaedics, Osaka University Graduate School of Medicine, Osaka, Japan.
Naomi
N
Yamasaki
Makoto
M
Hirao
Yoshitaka
Y
Kawato
Kosuke
K
Ebina
Hiroki
H
Oze
Akihide
A
Nampei
Kenrin
K
Shi
Hideki
H
Yoshikawa
Jun
J
Hashimoto
D000199:Actins / Q000235:genetics / Q000378:metabolism; D002135:Calcium-Binding Proteins / Q000235:genetics / Q000378:metabolism; D018929:Cell Culture Techniques; D015687:Cell Hypoxia; D002478:Cells, Cultured; D016326:Extracellular Matrix Proteins / Q000235:genetics / Q000378:metabolism; D015870:Gene Expression; D006801:Humans; D008840:Microfilament Proteins / Q000235:genetics / Q000378:metabolism; D009131:Muscle, Smooth, Vascular / Q000166:cytology*; D032389:Myocytes, Smooth Muscle / Q000378:metabolism* / Q000502:physiology; D015675:Osteocalcin / Q000235:genetics / Q000378:metabolism; D010641:Phenotype; D010750:Phosphoproteins / Q000235:genetics / Q000378:metabolism; D014841:von Willebrand Factor / Q000276:immunology / Q000378:metabolism; D000096985:Calponins
D000199:Actins; D002135:Calcium-Binding Proteins; D016326:Extracellular Matrix Proteins; D008840:Microfilament Proteins; D015675:Osteocalcin; D010750:Phosphoproteins; D014841:von Willebrand Factor; D000096985:Calponins; C513309:DMP1 protein, human; C029417:Von Willebrand antigen