title: Hypothesis-free analysis of ATG16L1 demonstrates gene-wide extent of association with Crohn's disease susceptibility.
authors: Johan
authors: J
authors: Van Limbergen
authors: Boyko
authors: B
authors: Kabakchiev
authors: Joanne M
authors: JM
authors: Stempak
authors: Phil
authors: P
authors: Schumm
authors: Wei
authors: W
authors: Xu
authors: Paul
authors: P
authors: Henderson
authors: Stephen
authors: S
authors: Girardin
authors: Anne M
authors: AM
authors: Griffiths
authors: Dana
authors: D
authors: Philpott
authors: Mark
authors: M
authors: Silverberg
mesh_terms: D002352:Carrier Proteins / Q000235:genetics*; D003424:Crohn Disease / Q000235:genetics*; D016207:Cytokines / Q000096:biosynthesis*; D006801:Humans; D018836:Inflammation Mediators / Q000378:metabolism*; D053473:Nod2 Signaling Adaptor Protein / Q000502:physiology*
chemical_list: D002352:Carrier Proteins; D016207:Cytokines; D018836:Inflammation Mediators; D053473:Nod2 Signaling Adaptor Protein