title: DNA hypermethylation promotes the low expression of pro-apoptotic BCL2L11 associated with BCR-ABL1 fusion gene of chronic myeloid leukaemia.
authors.forename: Elisa
authors.initials: E
authors.lastname: Leo
authors.forename: Manuela
authors.initials: M
authors.lastname: Mancini
authors.forename: Michela
authors.initials: M
authors.lastname: Aluigi
authors.forename: Fausto
authors.initials: F
authors.lastname: Castagnetti
authors.forename: Giovanni
authors.initials: G
authors.lastname: Martinelli
authors.forename: Enza
authors.initials: E
authors.lastname: Barbieri
authors.forename: Maria A
authors.initials: MA
authors.lastname: Santucci
mesh_terms: D017209:Apoptosis / Q000235:genetics*; D051017:Apoptosis Regulatory Proteins / Q000235:genetics*; D000072224:Bcl-2-Like Protein 11; D019175:DNA Methylation; D018450:Disease Progression; D019008:Drug Resistance, Neoplasm / Q000235:genetics; D016044:Fusion Proteins, bcr-abl / Q000235:genetics*; D015973:Gene Expression Regulation, Leukemic; D006801:Humans; D015464:Leukemia, Myelogenous, Chronic, BCR-ABL Positive / Q000235:genetics*; D008565:Membrane Proteins / Q000235:genetics*; D011401:Promoter Regions, Genetic; D011518:Proto-Oncogene Proteins / Q000235:genetics*
chemical_list: D051017:Apoptosis Regulatory Proteins; C000606775:BCL2L11 protein, human; D000072224:Bcl-2-Like Protein 11; D008565:Membrane Proteins; D011518:Proto-Oncogene Proteins; D016044:Fusion Proteins, bcr-abl