title: [Phenomenon of heart ischemic postconditioning].
abstract: Authors of review analyzed papers on problem of heart ischemic postconditioning. In the review, it was demonstrated that postconditioning decreased an infarct size, prevented cardiomyocytes apoptosis, improved cardiac contractility in reperfusion period, augmented cardiac tolerance to arrhythmogenic impact ofreperfusion, prevented neutrophil invasion into the reperfused heart, abolished reperfusion endothelial dysfunction and suppressed reperfusion oxidative stress in myocardium.
authors.forename: L N
authors.initials: LN
authors.lastname: Maslov
authors.forename: A G
authors.initials: AG
authors.lastname: Mrochek
authors.forename: L
authors.initials: L
authors.lastname: Hanus
authors.forename: J -M
authors.initials: J-
authors.lastname: Pei
authors.forename: Y
authors.initials: Y
authors.lastname: Zhang
authors.forename: H
authors.initials: H
authors.lastname: Wang
authors.forename: N V
authors.initials: NV
authors.lastname: Naryzhnaia
mesh_terms: D000818:Animals; D004730:Endothelium, Vascular / Q000378:metabolism* / Q000473:pathology; D006801:Humans; D019157:Ischemic Preconditioning, Myocardial / Q000379:methods*; D008297:Male; D015428:Myocardial Reperfusion Injury / Q000378:metabolism / Q000473:pathology / Q000517:prevention & control*; D009206:Myocardium / Q000378:metabolism* / Q000473:pathology; D018384:Oxidative Stress