title: Dentin hypersensitivity: pain mechanisms and aetiology of exposed cervical dentin.
abstract: OBJECTIVE
  The paper's aim is to review dentin hypersensitivity (DHS), discussing pain mechanisms and aetiology.
  
  
  METHODS
  Literature was reviewed using search engines with MESH terms, DH pain mechanisms and aetiology (including abrasion, erosion and periodontal disease).
  
  
  RESULTS
  The many hypotheses proposed for DHS attest to our lack of knowledge in understanding neurophysiologic mechanisms, the most widely accepted being the hydrodynamic theory. Dentin tubules must be patent from the oral environment to the pulp. Dentin exposure, usually at the cervical margin, is due to a variety of processes involving gingival recession or loss of enamel, predisposing factors being periodontal disease and treatment, limited alveolar bone, thin biotype, erosion and abrasion.
  
  
  CONCLUSIONS
  The current pain mechanism of DHS is thought to be the hydrodynamic theory. The initiation and progression of DHS are influenced by characteristics of the teeth and periodontium as well as the oral environment and external influences. Risk factors are numerous often acting synergistically and always influenced by individual susceptibility.
  
  
  CONCLUSIONS
  Whilst the pain mechanism of DHS is not well understood, clinicians need to be mindful of the aetiology and risk factors in order to manage patients' pain and expectations and prevent further dentin exposure with subsequent sensitivity.
authors.affiliation: Clinical Trials Unit, Department of Oral and Dental Sciences, Bristol Dental Hospital, Lower Maudlin Street, Bristol, BS1 2LY, UK. n.x.west@bristol.ac.uk
authors.forename: N X
authors.initials: NX
authors.lastname: West
authors.forename: A
authors.initials: A
authors.lastname: Lussi
authors.forename: J
authors.initials: J
authors.lastname: Seong
authors.forename: E
authors.initials: E
authors.lastname: Hellwig
mesh_terms: D003804:Dentin / Q000473:pathology; D003807:Dentin Sensitivity / Q000209:etiology / Q000503:physiopathology*; D004198:Disease Susceptibility / Q000503:physiopathology; D006801:Humans; D057446:Hydrodynamics; D010146:Pain / Q000503:physiopathology; D010510:Periodontal Diseases / Q000150:complications; D012307:Risk Factors; D019237:Tooth Cervix / Q000473:pathology; D057085:Tooth Wear / Q000150:complications