title: Initially disadvantaged, TEL-AML1 cells expand and initiate leukemia in response to irradiation and cooperating mutations.
authors: M
authors: M
authors: Li
authors: L
authors: L
authors: Jones
authors: C
authors: C
authors: Gaillard
authors: M
authors: M
authors: Binnewies
authors: R
authors: R
authors: Ochoa
authors: E
authors: E
authors: Garcia
authors: V
authors: V
authors: Lam
authors: G
authors: G
authors: Wei
authors: W
authors: W
authors: Yang
authors: C
authors: C
authors: Lobe
authors: M
authors: M
authors: Hermiston
authors: E
authors: E
authors: Passegué
authors: S C
authors: SC
authors: Kogan
mesh_terms: D000818:Animals; D050676:Core Binding Factor Alpha 2 Subunit / Q000235:genetics*; D004195:Disease Models, Animal; D049452:Green Fluorescent Proteins / Q000235:genetics; D006412:Hematopoietic Stem Cells / Q000187:drug effects* / Q000473:pathology; D006801:Humans; D051379:Mice; D008822:Mice, Transgenic; D009381:Neoplasms, Radiation-Induced / Q000473:pathology*; D015514:Oncogene Proteins, Fusion / Q000235:genetics*; D054198:Precursor Cell Lymphoblastic Leukemia-Lymphoma / Q000235:genetics / Q000473:pathology*; D000097764:ETS Translocation Variant 6 Protein
chemical_list: D050676:Core Binding Factor Alpha 2 Subunit; D049452:Green Fluorescent Proteins; D015514:Oncogene Proteins, Fusion; C102421:TEL-AML1 fusion protein; D000097764:ETS Translocation Variant 6 Protein; C485184:enhanced green fluorescent protein