title: Intravenous administration of Gr-1+CD11b+ myeloid cells increases neovascularization and improves cardiac function after heart infarction.
authors.affiliation: Key Laboratory of Medical Tissue Engineering of Liaoning Province, First Affiliated Hospital of Liaoning Medical University, Jinzhou, Liaoning, 121000, China; Department of Radiation Oncology, Vanderbilt University School of Medicine, Nashville, TN 37232, USA; Department of Surgical Laboratory, First Affiliated Hospital of Liaoning Medical University, Jinzhou, Liaoning, 121000, China. Electronic address: hjhdr@hotmail.com.
authors.forename: Jianhua
authors.initials: J
authors.lastname: Huang
authors.forename: Gang
authors.initials: G
authors.lastname: Lv
authors.forename: Yongfen
authors.initials: Y
authors.lastname: Min
authors.forename: Li
authors.initials: L
authors.lastname: Yang
authors.forename: P Charles
authors.initials: PC
authors.lastname: Lin
mesh_terms: D061605:Administration, Intravenous; D000818:Animals; D016026:Bone Marrow Transplantation / Q000379:methods; D039481:CD11b Antigen / Q000008:administration & dosage*; D051379:Mice; D008810:Mice, Inbred C57BL; D022423:Myeloid Cells / Q000637:transplantation*; D009203:Myocardial Infarction / Q000473:pathology / Q000601:surgery*; D018919:Neovascularization, Physiologic / Q000187:drug effects / Q000502:physiology*; D019707:Receptors, Chemokine / Q000008:administration & dosage*
chemical_list: D039481:CD11b Antigen; C503133:Gr-1 protein, mouse; D019707:Receptors, Chemokine
keywords: Angiogenesis; Cell therapy; Gr-1+CD11b+ myeloid cells; Heart infarction; Vasculogenesis