title: Adding a new facet to STAT5 in CML: multitasking for leukemic cells.
authors.forename: Wolfgang
authors.initials: W
authors.lastname: Warsch
authors.forename: Eva
authors.initials: E
authors.lastname: Grundschober
authors.forename: Veronika
authors.initials: V
authors.lastname: Sexl
mesh_terms: D000818:Animals; D016044:Fusion Proteins, bcr-abl / Q000235:genetics*; D006801:Humans; D015464:Leukemia, Myelogenous, Chronic, BCR-ABL Positive / Q000378:metabolism*; D009154:Mutation; D018384:Oxidative Stress; D011505:Protein-Tyrosine Kinases / Q000235:genetics*; D017382:Reactive Oxygen Species / Q000378:metabolism*; D050799:STAT5 Transcription Factor / Q000378:metabolism*; D025521:Tumor Suppressor Proteins / Q000378:metabolism*
chemical_list: D017382:Reactive Oxygen Species; D050799:STAT5 Transcription Factor; D025521:Tumor Suppressor Proteins; D011505:Protein-Tyrosine Kinases; D016044:Fusion Proteins, bcr-abl
keywords: ALL; BCR-ABL1; BCR-ABL1 mutation; CML; CML therapy; ROS; STAT5; TKI resistance; chronic myeloid leukemia