Medial and intimal calcification in chronic kidney disease: stressing the contributions.
Center for Interdisciplinary Cardiovascular Sciences, Division of Cardiovascular Medicine, Brigham and Women's Hospital, Harvard Medical School, Boston, MA.
Maximillian
M
Rogers
Claudia
C
Goettsch
Elena
E
Aikawa
D051701:Activating Transcription Factor 4 / Q000502:physiology*; D000818:Animals; D001011:Aorta / Q000378:metabolism*; D001018:Aortic Diseases / Q000209:etiology*; D059865:Endoplasmic Reticulum Stress / Q000502:physiology*; D006025:Glycosaminoglycans / Q000096:biosynthesis*; D008297:Male; D051436:Renal Insufficiency, Chronic / Q000150:complications* / Q000378:metabolism*; D051742:Transcription Factor CHOP / Q000502:physiology*; D014409:Tumor Necrosis Factor-alpha / Q000502:physiology*; D061205:Vascular Calcification / Q000209:etiology*; D019892:eIF-2 Kinase / Q000502:physiology*
D006025:Glycosaminoglycans; D014409:Tumor Necrosis Factor-alpha; D051701:Activating Transcription Factor 4; D051742:Transcription Factor CHOP; D019892:eIF-2 Kinase
Editorials; atherosclerosis; chronic kidney disease; endoplasmic reticulum stress; inflammation; intimal and medial calcification