Utilizing left ventricular outflow tract velocity changes to predict fluid responsiveness in septic patients: a case report.
Toxin-mediated vasodilation in the sepsis syndrome can lead to end-organ dysfunction and shock. Assessing for fluid responsiveness and preload optimization with intravenous fluids is a central tenet in the management of sepsis. Aggressive fluid administration can lead to pulmonary edema and heart failure, whereas premature inotropic or vasopressor support can worsen organ perfusion. Inferior vena cava ultrasonography is commonly used to assess for fluid responsiveness but has multiple limitations.
Department of Emergency Medicine, Olive View-UCLA Medical Center, Sylmar, CA. Electronic address: atchiem@gmail.com.
Alan T
AT
Chiem
Division of Pulmonary & Critical Care Medicine, Department of Medicine, Ronald Reagan-UCLA Medical Center, Los Angeles, CA.
Elizabeth
E
Turner
D000368:Aged; D018619:Echocardiography, Doppler, Pulsed; D005260:Female; D005440:Fluid Therapy; D006352:Heart Ventricles / Q000000981:diagnostic imaging*; D006801:Humans; D018805:Sepsis / Q000000981:diagnostic imaging / Q000503:physiopathology / Q000628:therapy*; D016277:Ventricular Function, Left