title: Unsuspected collision of synchronous lung adenocarcinomas: a potential cause of aberrant driver mutation profiles.
authors.affiliation: *Department of Pathology, †Human Oncology and Pathogenesis Program, Memorial Sloan-Kettering Cancer Center, New York, New York; ‡Computational Biology Program, Sloan-Kettering Institute, New York, New York; and §Department of Medicine Thoracic Oncology Service, Memorial Sloan-Kettering Cancer Center, New York, New York.
authors.forename: Natasha
authors.initials: N
authors.lastname: Rekhtman
authors.forename: Laetitia
authors.initials: L
authors.lastname: Borsu
authors.forename: Boris
authors.initials: B
authors.lastname: Reva
authors.forename: Maria
authors.initials: M
authors.lastname: Arcila
authors.forename: Gregory J
authors.initials: GJ
authors.lastname: Riely
authors.forename: Marc
authors.initials: M
authors.lastname: Ladanyi
authors.forename: Alexander
authors.initials: A
authors.lastname: Drilon
mesh_terms: D000230:Adenocarcinoma / Q000235:genetics*; D000077192:Adenocarcinoma of Lung; D000368:Aged; D005260:Female; D006801:Humans; D008175:Lung Neoplasms / Q000235:genetics*; D009154:Mutation; D009378:Neoplasms, Multiple Primary / Q000235:genetics*; D011518:Proto-Oncogene Proteins / Q000235:genetics*; D016283:Proto-Oncogene Proteins p21(ras); D018631:ras Proteins / Q000235:genetics*
chemical_list: C117307:KRAS protein, human; D011518:Proto-Oncogene Proteins; D016283:Proto-Oncogene Proteins p21(ras); D018631:ras Proteins