title: Could silencing IRF5 improve healing of a myocardial infarct through the reprogramming of the macrophage population?
authors.affiliation: Lillehei Heart Institute and Department of Medicine, University of Minnesota, Minneapolis, Minnesota. Electronic address: jlhall@umn.edu.
authors.forename: Jennifer L
authors.initials: JL
authors.lastname: Hall
authors.affiliation: Lillehei Heart Institute and Department of Pharmacology, University of Minnesota, Minneapolis, Minnesota.
authors.forename: Li-Na
authors.initials: LN
authors.lastname: Wei
mesh_terms: D000818:Animals; D005786:Gene Expression Regulation; D050835:Interferon Regulatory Factors / Q000235:genetics*; D008264:Macrophages / Q000378:metabolism*; D009203:Myocardial Infarction / Q000235:genetics*; D009206:Myocardium / Q000378:metabolism*; D012313:RNA / Q000235:genetics*; D020257:Ventricular Remodeling
chemical_list: D050835:Interferon Regulatory Factors; C498178:Irf5 protein, mouse; D012313:RNA
keywords: IRF5; M1 to M2 transition; macrophage; myocardial infarction