title: The battle in the gut.
abstract: Our molecular understanding of how pathogen-microbiota-immune system interactions influence disease outcomes is limited. In this issue of Immunity, Behnsen et al. (2014) report that the cytokine interleukin-22, which usually plays a protective role, promotes pathogen colonization by suppressing related commensal bacteria.
authors.affiliation: Department of Microbiology and Immunology, Stanford School of Medicine, Stanford University, Stanford, CA 94305, USA. Electronic address: dmonack@stanford.edu.
authors.forename: Denise M
authors.initials: DM
authors.lastname: Monack
mesh_terms: D000818:Animals; D054884:Host-Pathogen Interactions; D007378:Interleukins / Q000276:immunology*; D007422:Intestines / Q000382:microbiology*; D012480:Salmonella Infections / Q000276:immunology* / Q000382:microbiology*; D013559:Symbiosis / Q000276:immunology*; D000097043:Interleukin-22
chemical_list: D007378:Interleukins; D000097043:Interleukin-22