title: Arabidopsis ein2-1 and npr1-1 response to Al stress.
abstract: An enhanced Al(3+) tolerance has been observed in ethylene insensitive mutant ein2-1 and salicylic acid insensitive mutant npr1-1 of Arabidopsis. However, we found that the tolerant phenotype of ein2-1 and npr1-1 under Al stress was dependent on NPR and EIN function, respectively, because the double mutant ein2-1/npr1-1 displayed more sensitive to Al stress than wild-type plants. We analysed the differential performance between ein2-1/npr1-1 and their respective single mutant in response to Al stress, and found that antioxidant defence rather than malate exudation was the determinant factor.
authors.affiliation: College of Chemistry and Life Sciences, Shenyang Normal University, Shenyang, 110034, People's Republic of China.
authors.forename: Yiyan
authors.initials: Y
authors.lastname: Zhang
authors.forename: Qingqing
authors.initials: Q
authors.lastname: He
authors.forename: Shiyang
authors.initials: S
authors.lastname: Zhao
authors.forename: Linli
authors.initials: L
authors.lastname: Huang
authors.forename: Lin
authors.initials: L
authors.lastname: Hao
mesh_terms: D000535:Aluminum / Q000032:analysis / Q000378:metabolism / Q000633:toxicity*; D017360:Arabidopsis / Q000187:drug effects* / Q000235:genetics* / Q000254:growth & development / Q000378:metabolism; D029681:Arabidopsis Proteins / Q000235:genetics*; D002734:Chlorophyll / Q000032:analysis; D004789:Enzyme Activation / Q000187:drug effects; D018506:Gene Expression Regulation, Plant / Q000187:drug effects*; D009154:Mutation; D010088:Oxidoreductases / Q000378:metabolism; D018517:Plant Roots / Q000737:chemistry / Q000187:drug effects / Q000378:metabolism; D011956:Receptors, Cell Surface / Q000235:genetics*; D013312:Stress, Physiological / Q000187:drug effects* / Q000235:genetics
chemical_list: D029681:Arabidopsis Proteins; C119963:EIN2 protein, Arabidopsis; C105496:NPR1 protein, Arabidopsis; D011956:Receptors, Cell Surface; D002734:Chlorophyll; D000535:Aluminum; D010088:Oxidoreductases