title: Many BCR-ABL1 compound mutations reported in chronic myeloid leukemia patients may actually be artifacts due to PCR-mediated recombination.
authors.affiliation: Department of Genetics and Molecular Pathology, Centre for Cancer Biology, SA Pathology, Adelaide, Australia School of Pharmacy and Medical Science, University of South Australia, Adelaide, Australia.
authors.forename: Wendy T
authors.initials: WT
authors.lastname: Parker
authors.affiliation: Department of Genetics and Molecular Pathology, Centre for Cancer Biology, SA Pathology, Adelaide, Australia.
authors.forename: Stuart R
authors.initials: SR
authors.lastname: Phillis
authors.affiliation: Departments of Genetics and Molecular Pathology and Hematology, Centre for Cancer Biology, SA Pathology, Adelaide, AustraliaSchool of Medicine, University of Adelaide, Adelaide, Australia.
authors.forename: David T O
authors.initials: DT
authors.lastname: Yeung
authors.affiliation: Department of Hematology, Centre for Cancer Biology, SA Pathology, Adelaide, AustraliaSchool of Medicine, University of Adelaide, Adelaide, AustraliaCancer Theme, South Australian Health and Medical Research Institute, Adelaide, Australia.
authors.forename: Timothy P
authors.initials: TP
authors.lastname: Hughes
authors.affiliation: Department of Genetics and Molecular Pathology, Centre for Cancer Biology, SA Pathology, Adelaide, AustraliaSchools of Medicine and Molecular and Biomedical Science, University of Adelaide, Adelaide, AustraliaSchool of Pharmacy and Medical Science, University of South Australia, Adelaide, Australia.
authors.forename: Hamish S
authors.initials: HS
authors.lastname: Scott
authors.affiliation: Department of Genetics and Molecular Pathology, Centre for Cancer Biology, SA Pathology, Adelaide, AustraliaSchools of Medicine and Molecular and Biomedical Science, University of Adelaide, Adelaide, AustraliaSchool of Pharmacy and Medical Science, University of South Australia, Adelaide, Australia.
authors.forename: Susan
authors.initials: S
authors.lastname: Branford
mesh_terms: D016477:Artifacts; D016044:Fusion Proteins, bcr-abl / Q000235:genetics*; D006801:Humans; D015464:Leukemia, Myelogenous, Chronic, BCR-ABL Positive / Q000235:genetics*; D009154:Mutation; D016133:Polymerase Chain Reaction / Q000379:methods*
chemical_list: C581634:BCR-ABL1 fusion protein, human; D016044:Fusion Proteins, bcr-abl