title: Malaria parasite epigenetics: when virulence and romance collide.
abstract: Blood-stage malaria parasites evade the immune system by switching the protein exposed at the surface of the infected erythrocyte. A small proportion of these parasites commits to sexual development to mediate mosquito transmission. Two studies in this issue (Brancucci et al., 2014; Coleman et al., 2014) shed light on shared epigenetic machinery underlying both of these events.
authors.affiliation: Faculty of Infectious and Tropical Diseases, London School of Hygiene & Tropical Medicine, Keppel Street, London WC1E 7HT, UK. Electronic address: christian.flueck@lshtm.ac.uk.
authors.forename: Christian
authors.initials: C
authors.lastname: Flueck
authors.affiliation: Faculty of Infectious and Tropical Diseases, London School of Hygiene & Tropical Medicine, Keppel Street, London WC1E 7HT, UK. Electronic address: david.baker@lshtm.ac.uk.
authors.forename: David A
authors.initials: DA
authors.lastname: Baker
mesh_terms: D000953:Antigens, Protozoan / Q000276:immunology*; D000090266:Chromobox Protein Homolog 5; D002868:Chromosomal Proteins, Non-Histone / Q000502:physiology*; D006655:Histone Deacetylases / Q000502:physiology*; D006801:Humans; D016778:Malaria, Falciparum / Q000469:parasitology*; D010963:Plasmodium falciparum / Q000201:enzymology* / Q000502:physiology*; D015800:Protozoan Proteins / Q000502:physiology*
chemical_list: D000953:Antigens, Protozoan; D002868:Chromosomal Proteins, Non-Histone; D015800:Protozoan Proteins; D000090266:Chromobox Protein Homolog 5; D006655:Histone Deacetylases