title: Closing the gap between glia and neuroblast proliferation.
abstract: Reporting in this issue of Developmental Cell, Spéder and Brand (2014) show that gap junctions are required in blood-brain barrier glial cells to reactivate proliferation of quiescent neuroblasts. Gap junctions allow synchronous Ca(2+) waves and control insulin-like protein Dipl6 expression and secretion to trigger neuroblast division.
authors.affiliation: Institut für Neuro- und Verhaltensbiologie, Badestrasse 9, 48149 Münster, Germany.
authors.forename: Stefanie
authors.initials: S
authors.lastname: Limmer
authors.affiliation: Institut für Neuro- und Verhaltensbiologie, Badestrasse 9, 48149 Münster, Germany. Electronic address: klaembt@uni-muenster.de.
authors.forename: Christian
authors.initials: C
authors.lastname: Klämbt
mesh_terms: D000818:Animals; D001812:Blood-Brain Barrier / Q000378:metabolism; D001921:Brain / Q000378:metabolism; D017630:Connexins / Q000378:metabolism; D029721:Drosophila Proteins / Q000378:metabolism; D058953:Neural Stem Cells / Q000378:metabolism; D015398:Signal Transduction / Q000502:physiology
chemical_list: D017630:Connexins; D029721:Drosophila Proteins