title: Introduction: Are we advancing in our scientific understanding and therapeutic improvement of uterine fibroids… or not?
abstract: The cellular, genetic, and hormonal understanding of uterine fibroids is rapidly growing, but the therapeutic options remain similar. Are we really advancing the field?
authors.affiliation: Fundación Instituto Valenciano de Infertilidad, Instituto Universitario IVI, University of Valencia, INCLIVA, Valencia, Spain; and Department of Obstetrics and Gynecology, School of Medicine, Stanford University, Stanford, California. Electronic address: jaime.herrero@iviomics.com.
authors.forename: Carlos
authors.initials: C
authors.lastname: Simón
mesh_terms: D020847:Estrogen Receptor Modulators / Q000627:therapeutic use; D005260:Female; D025743:HMGA2 Protein / Q000235:genetics; D006801:Humans; D015994:Incidence; D007889:Leiomyoma / Q000453:epidemiology / Q000209:etiology* / Q000628:therapy*; D056892:Mediator Complex / Q000235:genetics; D011987:Receptors, Steroid / Q000235:genetics; D012307:Risk Factors; D015398:Signal Transduction / Q000235:genetics; D014594:Uterine Neoplasms / Q000453:epidemiology / Q000209:etiology* / Q000628:therapy*
chemical_list: D020847:Estrogen Receptor Modulators; D025743:HMGA2 Protein; C460107:MED12 protein, human; D056892:Mediator Complex; D011987:Receptors, Steroid
keywords: MED12; Uterine fibroids; myomas; stem cells; treatment