title: Endothelial dysfunction potentially interacts with impaired glucose metabolism to increase cardiovascular risk.
authors.affiliation: From the Division of Endocrinology, Diabetes, and Metabolism, Department of Medicine, Diabetes Cardiovascular Center (G.J., J.R.S.), and Departments of Medical Pharmacology and Physiology (J.R.S.), University of Missouri, Columbia; and Harry S. Truman Memorial Veterans Hospital (G.J., J.R.S.), Columbia, MO. jiag@health.missouri.edu.
authors.forename: Guanghong
authors.initials: G
authors.lastname: Jia
authors.affiliation: From the Division of Endocrinology, Diabetes, and Metabolism, Department of Medicine, Diabetes Cardiovascular Center (G.J., J.R.S.), and Departments of Medical Pharmacology and Physiology (J.R.S.), University of Missouri, Columbia; and Harry S. Truman Memorial Veterans Hospital (G.J., J.R.S.), Columbia, MO.
authors.forename: James R
authors.initials: JR
authors.lastname: Sowers
mesh_terms: D002318:Cardiovascular Diseases / Q000503:physiopathology*; D003924:Diabetes Mellitus, Type 2 / Q000150:complications*; D004730:Endothelium, Vascular / Q000503:physiopathology*; D005260:Female; D006801:Humans; D008297:Male; D011159:Population Surveillance / Q000379:methods*; D014655:Vascular Resistance / Q000502:physiology*; D014664:Vasodilation / Q000502:physiology*
keywords: cardiovascular disease; population; type 2 diabetes mellitus