title: ALT telomeres borrow from meiosis to get moving.
abstract: Telomere clustering is required for the homologous recombination events that maintain chromosome ends in cells relying on alternative lengthening of telomeres (ALT). New data demonstrate that damage signaling at telomeres, a likely step in activating maintenance mechanisms, induces directional movement and synapsis driven by the machinery responsible for recombination in meiosis.
authors.affiliation: The Salk Institute for Biological Studies, Molecular and Cell Biology Laboratory, 10010 North Torrey Pines Road, La Jolla, CA 92037, USA.
authors.forename: Nausica
authors.initials: N
authors.lastname: Arnoult
authors.affiliation: The Salk Institute for Biological Studies, Molecular and Cell Biology Laboratory, 10010 North Torrey Pines Road, La Jolla, CA 92037, USA. Electronic address: karlseder@salk.edu.
authors.forename: Jan
authors.initials: J
authors.lastname: Karlseder
mesh_terms: D023902:Chromosome Pairing; D006801:Humans; D011995:Recombination, Genetic; D016615:Telomere / Q000378:metabolism*