title: Stress heals.
abstract: Reactive oxygen species (ROS) are generated as a response to cellular stress and regulate processes including cellular signaling and wound healing. In this issue of Developmental Cell, Xu and Chisholm (2014) demonstrate that mitochondrial ROS are required for proper wound healing in Caenorhabditis elegans through controlling the redox state of actin regulators.
authors.affiliation: Cell Biology Program, Memorial Sloan-Kettering Cancer Center, New York, NY 10065, USA. Electronic address: niethamp@mskcc.org.
authors.forename: Philipp
authors.initials: P
authors.lastname: Niethammer
mesh_terms: D000818:Animals; D017173:Caenorhabditis elegans / Q000378:metabolism*; D029742:Caenorhabditis elegans Proteins / Q000378:metabolism*; D004817:Epidermis / Q000378:metabolism*; D008928:Mitochondria / Q000378:metabolism*; D033681:Mitochondrial Membrane Transport Proteins / Q000378:metabolism*; D017382:Reactive Oxygen Species / Q000378:metabolism*; D014945:Wound Healing
chemical_list: D029742:Caenorhabditis elegans Proteins; C000594125:Mcu-1 protein, C elegans; D033681:Mitochondrial Membrane Transport Proteins; D017382:Reactive Oxygen Species