title: Hypoxia in plaque macrophages: a new danger signal for interleukin-1β activation?
authors.affiliation: From the Cardiovascular Division, Department of Medicine (I.S., T.D.E., S.B., B.R.) and Department of Pathology and Immunology (B.R.), Washington University School of Medicine, St. Louis, MO.
authors.forename: Ismail
authors.initials: I
authors.lastname: Sergin
authors.affiliation: From the Cardiovascular Division, Department of Medicine (I.S., T.D.E., S.B., B.R.) and Department of Pathology and Immunology (B.R.), Washington University School of Medicine, St. Louis, MO.
authors.forename: Trent D
authors.initials: TD
authors.lastname: Evans
authors.affiliation: From the Cardiovascular Division, Department of Medicine (I.S., T.D.E., S.B., B.R.) and Department of Pathology and Immunology (B.R.), Washington University School of Medicine, St. Louis, MO.
authors.forename: Somashubhra
authors.initials: S
authors.lastname: Bhattacharya
authors.affiliation: From the Cardiovascular Division, Department of Medicine (I.S., T.D.E., S.B., B.R.) and Department of Pathology and Immunology (B.R.), Washington University School of Medicine, St. Louis, MO. brazani@im.wustl.edu.
authors.forename: Babak
authors.initials: B
authors.lastname: Razani
mesh_terms: D006801:Humans; D053583:Interleukin-1beta / Q000096:biosynthesis*; D008264:Macrophages / Q000378:metabolism*
chemical_list: D053583:Interleukin-1beta
keywords: Editorials; atherosclerosis; autophagy; cellular hypoxia; inflammation; interleukins; macrophages