title: Comment on Sato et al. Improving type 2 diabetes through a distinct adrenergic signaling pathway involving mTORC2 that mediates glucose uptake in skeletal muscle. Diabetes 2014;63:4115-4129.
authors.affiliation: Department of Physiology and Biophysics, University of Mississippi Medical Center, Jackson, MS lxiang2@umc.edu.
authors.forename: Lusha
authors.initials: L
authors.lastname: Xiang
authors.affiliation: Department of Physiology and Biophysics, University of Mississippi Medical Center, Jackson, MS.
authors.forename: Peter N
authors.initials: PN
authors.lastname: Mittwede
authors.affiliation: Department of Physiology and Biophysics, University of Mississippi Medical Center, Jackson, MS.
authors.forename: Robert L
authors.initials: RL
authors.lastname: Hester
mesh_terms: D000818:Animals; D001786:Blood Glucose / Q000378:metabolism*; D003921:Diabetes Mellitus, Experimental / Q000378:metabolism*; D003924:Diabetes Mellitus, Type 2 / Q000378:metabolism*; D051275:Glucose Transporter Type 4 / Q000378:metabolism*; D000076225:Mechanistic Target of Rapamycin Complex 2; D046912:Multiprotein Complexes / Q000378:metabolism*; D018482:Muscle, Skeletal / Q000378:metabolism*; D018343:Receptors, Adrenergic, beta-2 / Q000378:metabolism*; D058570:TOR Serine-Threonine Kinases / Q000378:metabolism*
chemical_list: D001786:Blood Glucose; D051275:Glucose Transporter Type 4; D046912:Multiprotein Complexes; D018343:Receptors, Adrenergic, beta-2; D000076225:Mechanistic Target of Rapamycin Complex 2; D058570:TOR Serine-Threonine Kinases