title: "Toll"-erance in the skin.
abstract: Interactions between potentially pathogenic commensal bacteria and cutaneous immunity are poorly understood. In this issue of Immunity, Skabytska et al. (2014) show that S. aureus-derived TLR2/6 heterodimer ligands can recruit myeloid-derived suppressor cells into the skin, countering rather than promoting inflammation.
authors.affiliation: Department of Dermatology, University of Pittsburgh School of Medicine, Pittsburgh, PA 15213, USA.
authors.forename: Tina L
authors.initials: TL
authors.lastname: Sumpter
authors.affiliation: Department of Dermatology, University of Pittsburgh School of Medicine, Pittsburgh, PA 15213, USA; Department of Bioengineering, University of Pittsburgh School of Medicine, Pittsburgh, PA 15213, USA; Pittsburgh Clinical and Translational Science Institute, University of Pittsburgh School of Medicine, Pittsburgh, PA 15213, USA; The McGowan Institute for Regenerative Medicine, University of Pittsburgh School of Medicine, Pittsburgh, PA 15213, USA; The University of Pittsburgh Cancer Institute, University of Pittsburgh School of Medicine, Pittsburgh, PA 15213, USA. Electronic address: lof2@pitt.edu.
authors.forename: Louis D
authors.initials: LD
authors.lastname: Falo
mesh_terms: D000818:Animals; D006801:Humans; D022423:Myeloid Cells / Q000276:immunology*; D012867:Skin / Q000276:immunology*; D013207:Staphylococcal Skin Infections / Q000276:immunology*; D051195:Toll-Like Receptor 2 / Q000276:immunology*
chemical_list: C493486:Tlr2 protein, mouse; D051195:Toll-Like Receptor 2