title: Why we can talk, debate, and change our minds: neural circuits, basal ganglia operations, and transcriptional factors.
abstract: Ackermann et al. disregard attested knowledge concerning aphasia, Parkinson disease, cortical-to-striatal circuits, basal ganglia, laryngeal phonation, and other matters. Their dual-pathway model cannot account for "what is special about the human brain." Their human cortical-to-laryngeal neural circuit does not exist. Basal ganglia operations, enhanced by mutations on FOXP2, confer human motor-control, linguistic, and cognitive capabilities.
authors.affiliation: Department of Cognitive,Linguistic and Psychological Sciences,Brown University,Providence,RI 02912.Philip.Lieberman@gmail.comwww.Brown.edu.
authors.forename: Philip
authors.initials: P
authors.lastname: Lieberman
mesh_terms: D000819:Animal Communication; D000818:Animals; D005075:Biological Evolution; D003142:Communication; D006801:Humans; D011323:Primates / Q000502:physiology*; D013060:Speech / Q000502:physiology*