title: Myeloid cells stimulate their progenitors in an emergency.
abstract: The nature and functions of cells creating hematopoietic niches during inflammation remain incompletely understood. In this issue of Immunity, Kwak et al. (2015) reveal that myeloid cell-produced reactive oxygen species stimulate proliferation of myeloid progenitors establishing an additional mechanism to regulate hematopoiesis.
authors.affiliation: Department of Immunobiology and Hematology, Institute for Frontier Medical Sciences, Kyoto University, Kyoto 606-8507, Japan; Japan Science and Technology Agency (JST), Core Research for Evolutional Science and Technology (CREST), 53 Kawahara-cho, Shogoin, Sakyo-ku, Kyoto 606-8507, Japan.
authors.forename: Tatsuki
authors.initials: T
authors.lastname: Sugiyama
authors.affiliation: Department of Immunobiology and Hematology, Institute for Frontier Medical Sciences, Kyoto University, Kyoto 606-8507, Japan; Japan Science and Technology Agency (JST), Core Research for Evolutional Science and Technology (CREST), 53 Kawahara-cho, Shogoin, Sakyo-ku, Kyoto 606-8507, Japan. Electronic address: tnagasa@frontier.kyoto-u.ac.jp.
authors.forename: Takashi
authors.initials: T
authors.lastname: Nagasawa
mesh_terms: D000818:Animals; D004926:Escherichia coli / Q000276:immunology*; D004927:Escherichia coli Infections / Q000276:immunology*; D006098:Granulocytes / Q000502:physiology*; D006410:Hematopoiesis; D022423:Myeloid Cells / Q000502:physiology*; D023461:Myeloid Progenitor Cells / Q000502:physiology*