Can the DNA damage response be harnessed to modulate atherosclerotic plaque phenotype?
From the Cardiovascular Division, Departments of Medicine and Pathology/Immunology, Washington University School of Medicine, St. Louis, MO (B.R.); and Department of Pathology, University of Washington School of Medicine, Seattle (E.W.R.). brazani@im.wustl.edu ewraines@uw.edu.
Babak
B
Razani
From the Cardiovascular Division, Departments of Medicine and Pathology/Immunology, Washington University School of Medicine, St. Louis, MO (B.R.); and Department of Pathology, University of Washington School of Medicine, Seattle (E.W.R.). brazani@im.wustl.edu ewraines@uw.edu.
Elaine W
EW
Raines
D000818:Animals; D004249:DNA Damage; D005260:Female; D006801:Humans; D008297:Male; D009131:Muscle, Smooth, Vascular / Q000378:metabolism*; D032389:Myocytes, Smooth Muscle / Q000378:metabolism*; D058226:Plaque, Atherosclerotic / Q000235:genetics*
DNA breaks, double-stranded; Editorials; myocytes, smooth muscle