title: Loss of HtrA1-induced attenuation of TGF-β signaling in fibroblasts might not be the main mechanism of CARASIL pathogenesis.
authors.affiliation: Medical Research Center, Shandong Provincial Qianfoshan Hospital, Shandong University, Jinan, Shandong 250014, China; and Department of Epidemiology and Public Health, Yale University, New Haven, CT 06520 ju.liu@sdu.edu.cn.
authors.forename: Ju
authors.initials: J
authors.lastname: Liu
authors.affiliation: Medical Research Center, Shandong Provincial Qianfoshan Hospital, Shandong University, Jinan, Shandong 250014, China; and.
authors.forename: Fengyun
authors.initials: F
authors.lastname: Dong
authors.affiliation: Department of Epidemiology and Public Health, Yale University, New Haven, CT 06520.
authors.forename: Josephine
authors.initials: J
authors.lastname: Hoh
mesh_terms: D000505:Alopecia / Q000235:genetics*; D000818:Animals; D002544:Cerebral Infarction / Q000235:genetics*; D006801:Humans; D053779:Latent TGF-beta Binding Proteins / Q000502:physiology*; D056784:Leukoencephalopathies / Q000235:genetics*; D012697:Serine Endopeptidases / Q000502:physiology*; D013122:Spinal Diseases / Q000235:genetics*; D053773:Transforming Growth Factor beta1 / Q000502:physiology*
chemical_list: D053779:Latent TGF-beta Binding Proteins; D053773:Transforming Growth Factor beta1; D012697:Serine Endopeptidases