title: Inhibition of hypertrophy, per se, may not be a good therapeutic strategy in ventricular pressure overload: other approaches could be more beneficial.
authors.affiliation: From Université Paris-Sud 11, and Institut National de la Santé et de la Recherche Médicale, Unit 1180, Châtenay-Malabry, France. Bertrand.crozatier@u-psud.fr.
authors.forename: Bertrand
authors.initials: B
authors.lastname: Crozatier
authors.affiliation: From Université Paris-Sud 11, and Institut National de la Santé et de la Recherche Médicale, Unit 1180, Châtenay-Malabry, France.
authors.forename: Renée
authors.initials: R
authors.lastname: Ventura-Clapier
mesh_terms: D000818:Animals; D001024:Aortic Valve Stenosis / Q000150:complications; D065095:Calcineurin Inhibitors / Q000627:therapeutic use / Q000633:toxicity; D003326:Coronary Circulation; D004730:Endothelium, Vascular / Q000473:pathology; D005260:Female; D005355:Fibrosis; D056572:Histone Deacetylase Inhibitors / Q000627:therapeutic use; D006801:Humans; D017379:Hypertrophy, Left Ventricular / Q000503:physiopathology / Q000628:therapy*; D008297:Male; D009200:Myocardial Contraction / Q000187:drug effects / Q000502:physiology; D032383:Myocytes, Cardiac / Q000378:metabolism / Q000473:pathology*; D009389:Neovascularization, Pathologic / Q000503:physiopathology; D012737:Sex Factors; D015398:Signal Transduction / Q000187:drug effects; D020257:Ventricular Remodeling
chemical_list: D065095:Calcineurin Inhibitors; D056572:Histone Deacetylase Inhibitors