title: Inhibition of hypertrophy is a good therapeutic strategy in ventricular pressure overload.
authors.affiliation: From Departments of Internal Medicine (Cardiology) (G.G.S., J.A.H.) and Molecular Biology (J.A.H.), University of Texas Southwestern Medical Center, Dallas, TX.
authors.forename: Gabriele G
authors.initials: GG
authors.lastname: Schiattarella
authors.affiliation: From Departments of Internal Medicine (Cardiology) (G.G.S., J.A.H.) and Molecular Biology (J.A.H.), University of Texas Southwestern Medical Center, Dallas, TX. joseph.hill@utsouthwestern.edu.
authors.forename: Joseph A
authors.initials: JA
authors.lastname: Hill
mesh_terms: D000818:Animals; D016923:Cell Death; D004730:Endothelium, Vascular / Q000473:pathology; D005355:Fibrosis; D005786:Gene Expression Regulation; D006329:Heart Conduction System / Q000503:physiopathology; D056572:Histone Deacetylase Inhibitors / Q000627:therapeutic use; D006801:Humans; D006973:Hypertension / Q000150:complications / Q000188:drug therapy; D017379:Hypertrophy, Left Ventricular / Q000503:physiopathology / Q000517:prevention & control*; D008264:Macrophages / Q000378:metabolism; D032383:Myocytes, Cardiac / Q000378:metabolism / Q000473:pathology*; D020257:Ventricular Remodeling
chemical_list: D056572:Histone Deacetylase Inhibitors