Somatostatin receptor subtype 2 as pancreatic tumorigenesis suppressor: identification of a new targetable signaling node.
Departments of Molecular & Integrative Physiology and Internal Medicine, University of Michigan, Ann Arbor, Michigan. Electronic address: howcraw@umich.edu.
Howard C
HC
Crawford
D000818:Animals; D021441:Carcinoma, Pancreatic Ductal / Q000201:enzymology*; D049109:Cell Proliferation; D054427:Chemokine CXCL6 / Q000378:metabolism*; D006801:Humans; D009154:Mutation; D010190:Pancreatic Neoplasms / Q000201:enzymology*; D058539:Phosphatidylinositol 3-Kinase / Q000378:metabolism*; D016283:Proto-Oncogene Proteins p21(ras) / Q000235:genetics*; D017481:Receptors, Somatostatin / Q000172:deficiency*; D015398:Signal Transduction
D054427:Chemokine CXCL6; D017481:Receptors, Somatostatin; D058539:Phosphatidylinositol 3-Kinase; D016283:Proto-Oncogene Proteins p21(ras)