Wall-lesion development in gaps: The role of the adhesive bonding material.
OBJECTIVE
This study evaluated the caries wall lesion development in different composite-dentin interfaces to investigate if the presence and location of two bonding materials in the gaps influence wall caries lesion development.


METHODS
Fourteen volunteers wore a modified occlusal splint containing samples with four different interfaces: perfect bonding/no gap, or with a fixed gap (234±30 μm) with either no bonding material, bonding material (Clearfil Protect Bond-PB and Clearfil SE Bond-SE) on dentin or on composite. Eight times a day, the samples were dipped in 20% sucrose solution for 10 min, during 3 weeks. The samples were imaged with microradiography (T-WIM), and lesion depth (LD) and mineral loss (ML) were measured. The data were analysed with paired t-test.


RESULTS
The perfect bonding group did not show any caries wall lesion development, whereas all other interfaces did. The interface with bonding on dentin did not show significantly different wall lesion development from the interface with no material. However, when bonding was present on composite, both LD and ML were significantly higher than both other gap conditions (p-values<0.05). A difference between the bonding material was only seen when applied on composite: PB showed less ML than SE (p=0.01).


CONCLUSIONS
The presence of bonding on the composite side of a composite-dentin gap increased wall lesion development in situ.


CONCLUSIONS
The presence and location of an adhesive bonding material in the composite-dentin gaps plays a role on the wall caries lesion development.
School of Dentistry, Federal University of Pelotas, Department of Restorative Dentistry, Pelotas, Brazil; Radboud University Medical Center, Radboud Institute for Health Sciences, Department of Dentistry, Nijmegen, The Netherlands. Electronic address: animontag@gmail.com.
Anelise F
AF
Montagner
Radboud University Medical Center, Radboud Institute for Health Sciences, Department of Dentistry, Nijmegen, The Netherlands. Electronic address: nicolien.kuper@radboudumc.nl.
Nicolien K
NK
Kuper
Radboud University Medical Center, Radboud Institute for Health Sciences, Department of Dentistry, Nijmegen, The Netherlands. Electronic address: niek.opdam@radboudumc.nl.
Niek J M
NJ
Opdam
Radboud University Medical Center, Radboud Institute for Health Sciences, Department of Dentistry, Nijmegen, The Netherlands. Electronic address: ewald.bronkhorst@radboudumc.nl.
Ewald M
EM
Bronkhorst
School of Dentistry, Federal University of Pelotas, Department of Restorative Dentistry, Pelotas, Brazil. Electronic address: cencims@gmail.com.
Maximiliano S
MS
Cenci
Radboud University Medical Center, Radboud Institute for Health Sciences, Department of Dentistry, Nijmegen, The Netherlands. Electronic address: marie-charlotte.huysmans@radboudumc.nl.
Marie-Charlotte D N J M
MC
Huysmans
D000293:Adolescent; D000328:Adult; D001862:Bone Resorption; D003731:Dental Caries / Q000139:chemically induced* / Q000473:pathology*; D003738:Dental Cements / Q000378:metabolism*; D003804:Dentin / Q000378:metabolism* / Q000473:pathology*; D005260:Female; D006801:Humans; D008297:Male; D008875:Middle Aged; D014838:Volunteers; D055815:Young Adult
D003738:Dental Cements
Adhesive system; Caries; Demineralization; Dental materials; Dentin; Mineral loss; Secondary caries; T-WIM.