title: Shedding light on glioma growth.
abstract: Cancer is known for opportunistically utilizing resources from its surroundings for its own growth and survival. In this issue of Cell, Venkatesh et al. demonstrate that this also occurs in the brain, identifying neuronal activity-induced secretion of neuroligin-3 as a novel mechanism promoting glioma proliferation.
authors.affiliation: Department of Neurology, F.M. Kirby Neurobiology Center, Boston Children's Hospital, Harvard Medical School, Boston, MA 02115, USA.
authors.forename: Emily K
authors.initials: EK
authors.lastname: Lehrman
authors.affiliation: Department of Neurology, F.M. Kirby Neurobiology Center, Boston Children's Hospital, Harvard Medical School, Boston, MA 02115, USA. Electronic address: beth.stevens@childrens.harvard.edu.
authors.forename: Beth
authors.initials: B
authors.lastname: Stevens
mesh_terms: D000818:Animals; D001932:Brain Neoplasms / Q000473:pathology*; D015816:Cell Adhesion Molecules, Neuronal / Q000378:metabolism*; D049109:Cell Proliferation; D005910:Glioma / Q000473:pathology*; D006801:Humans; D008297:Male; D008565:Membrane Proteins / Q000378:metabolism*; D009419:Nerve Tissue Proteins / Q000378:metabolism*
chemical_list: D015816:Cell Adhesion Molecules, Neuronal; D008565:Membrane Proteins; D009419:Nerve Tissue Proteins; C098004:neuroligin 3