title: [FEATURES IMMUNITY IN CHILDREN WITH BRONCHOPULMONARY DYSPLASIA].
abstract: The study of the immune system in 131 children with bronchopulmonary dysplasia at the time of remission and exacerbation. Found that children with bronchopulmonary dysplasia inherent low absolute number of lymphocytes, accompanied by increased cytotoxic lymphocytes (CD8, CD 16), oxygendependence ability of granulocytes, and activation of the acute phase of immunoglobulin (IgM), in the absence of clinical signs of exacerbation. In acute respiratory disease, a somewhat paradoxical, cellular immune response with decreased lymphocyte activation against cytotoxic cells. While the dynamics of increase of cytotoxic markers in acute respiratory disease is unsatisfactory, indicating that the depletion of cellular reactivity circuit immunity.
authors.forename: H S
authors.initials: HS
authors.lastname: Senatorova
authors.forename: O L
authors.initials: OL
authors.lastname: Lohvinova
mesh_terms: D001997:Bronchopulmonary Dysplasia / Q000097:blood* / Q000276:immunology*; D016827:CD8 Antigens / Q000276:immunology; D016022:Case-Control Studies; D002675:Child, Preschool; D003627:Data Interpretation, Statistical; D058851:GPI-Linked Proteins / Q000276:immunology; D006801:Humans; D007111:Immunity, Cellular; D056724:Immunity, Humoral; D007075:Immunoglobulin M / Q000097:blood; D007223:Infant; D018655:Lymphocyte Count; D008214:Lymphocytes / Q000166:cytology / Q000276:immunology*; D010587:Phagocytosis / Q000276:immunology; D011237:Predictive Value of Tests; D017452:Receptors, IgG / Q000276:immunology; D013602:T-Lymphocytes, Cytotoxic / Q000166:cytology / Q000276:immunology
chemical_list: D016827:CD8 Antigens; C428200:FCGR3B protein, human; D058851:GPI-Linked Proteins; D007075:Immunoglobulin M; D017452:Receptors, IgG