title: β3-Adrenoceptor, glutathionylation, and diabetic cardiomyopathy. Focus on "β3-Adrenoceptor activation relieves oxidative inhibition of the cardiac Na+-K+ pump in hyperglycemia induced by insulin receptor blockade".
authors.affiliation: Division of Translational and Regenerative Medicine, Department of Medicine, University of Arizona, Tucson, Arizona steveblack@email.arizona.edu.
authors.forename: Stephen M
authors.initials: SM
authors.lastname: Black
mesh_terms: D000318:Adrenergic beta-Agonists / Q000008:administration & dosage; D000818:Animals; D058065:Diabetic Cardiomyopathies / Q000188:drug therapy / Q000378:metabolism*; D016503:Drug Delivery Systems / Q000639:trends; D005978:Glutathione / Q000378:metabolism; D006801:Humans; D006943:Hyperglycemia / Q000188:drug therapy / Q000378:metabolism*; D018384:Oxidative Stress / Q000187:drug effects / Q000502:physiology*; D011972:Receptor, Insulin / Q000037:antagonists & inhibitors*; D022702:Receptors, Adrenergic, beta-3 / Q000378:metabolism*; D000254:Sodium-Potassium-Exchanging ATPase / Q000037:antagonists & inhibitors*
chemical_list: D000318:Adrenergic beta-Agonists; D022702:Receptors, Adrenergic, beta-3; D011972:Receptor, Insulin; D000254:Sodium-Potassium-Exchanging ATPase; D005978:Glutathione