Malformations of the Left Ventricle: What Comes First: Form or Function?
From the Department of Pediatric Cardiology, Charité-University Medicine Berlin & Experimental and Clinical Research Center, a joint cooperation between the Charité Medical Faculty and the Max-Delbrück-Center for Molecular Medicine, Berlin, Germany. klaassen@mdc-berlin.de.
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Klaassen
D000818:Animals; D024722:Cardiac Myosins / Q000235:genetics*; D005260:Female; D016368:Frameshift Mutation; D020022:Genetic Predisposition to Disease / Q000235:genetics*; D006801:Humans; D018636:Hypoplastic Left Heart Syndrome / Q000235:genetics*; D056830:Isolated Noncompaction of the Ventricular Myocardium / Q000235:genetics*; D008297:Male; D009154:Mutation; D018995:Myosin Heavy Chains / Q000235:genetics*; D061087:NADP Transhydrogenase, AB-Specific / Q000235:genetics*; D013318:Stroke Volume / Q000235:genetics*
D061087:NADP Transhydrogenase, AB-Specific; D024722:Cardiac Myosins; D018995:Myosin Heavy Chains
Editorials; cardiomyopathy; congenital heart disease; noncompaction cardiomyopathy; sarcomere